Description |
Dehydroascorbic acid, a blood-brain barrier transportable form of vitamin C, mediates potent cerebroprotection in experimental stroke.
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Target |
Human Endogenous Metabolite
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In Vitro |
Neuronal injury in ischemic stroke is partly mediated by cytotoxic reactive oxygen species. Although the antioxidant ascorbic acid (AA) or vitamin C does not penetrate the blood-brain barrier (BBB), its oxidized form, dehydroascorbic acid (DHA), enters the brain by means of facilitative transport[1].
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Density | 1.7±0.1 g/cm3 |
Boiling Point | 389.3±38.0 °C at 760 mmHg |
Flash Point | 170.0±20.3 °C |
Exact Mass | 174.016434 |
PSA | 100.90000 |
LogP | -2.32 |
Vapour Pressure | 0.0±2.0 mmHg at 25°C |
Storage condition | −20°C |